Left kidney hydronephrosis means that the left urethra is blocked, which causes the urine in the left kidney to be discharged poorly. If hydronephrosis occurs, you need to see what causes it. Generally speaking, the most common cause of hydronephrosis is ureteral stones, which are usually accompanied by lower back and abdominal pain, nausea, and vomiting. In addition, ureteral stenosis, cystic edema, and compression of retroperitoneal tumors can all cause hydronephrosis. It is best to have a urogenital system angiography to find out the cause of hydronephrosis and then use medication to treat it. As urine accumulates in the kidney, working pressure increases, causing the renal pelvis and calyx to expand and the renal parenchyma to shrink. If the retained urine becomes infected, it is called rational hydronephrosis; when the renal tissue necrotizes and loses its function due to infection, the renal pelvis is filled with concentrated fluid, which is called nephrotic or purulent kidney. The most critical cause of hydronephrosis is obstruction of the junction of the urethra and pelvis. 1. Congenital causes of infarction: Segmental hypotension: Due to segmental muscle deficiency, dysplasia or anatomical disorder at the junction of the urethral tube or the upper urethra, the normal peristalsis of this section of the urethra is affected, resulting in obstruction of driving force. If such changes occur at the bladder entrance of the urethra, congenital megaurethra will be produced, with adverse effects such as dilation and water retention of the kidney and ureter. Intrinsic ureteral stenosis: Most cases occur at the junction of the urethral pelvis. The narrow segment is usually 1-2 mm, but can be as long as 1-3 cm, causing incomplete obstruction and secondary distortion. Under transmission electron microscopy, it can be seen that there are too many fiber cells around the muscle cells and in the middle of the body cells in the obstructed segment. Over time, the muscle cells are damaged, resulting in an inelastic narrow segment dominated by fiber cells, which blocks the transmission of urine and causes hydronephrosis. To supplement the urethral tube distortion, adhesion, bandage or heart valve prong structure, which can be congenital or acquired, often occurs at the junction of the urethral tube and the lumbar section of the urethra, and basically accounts for 2/3 of adolescents and babies. The heavy disordered blood vessels are compressed by about 1/3, which are disordered hilar blood vessels, located in front of the junction of the renal pelvis and urethra. Others include horseshoe-shaped kidneys and obstruction of kidney function rotation during embryonic development. Superior urethral opening at term: It may be congenital or may be caused by asymptomatic pelvic dilatation due to peripheral pelvic fibrosis or bladder urethral reflux, resulting in a relative upward shift of the pelvic urethral junction, which cannot be detected during surgery. Congenital urethral disorder, cystic ulcer, double urethral tube, etc. |
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