After ejaculating once, it is not so hard anymore

There is a limit to the amount of sperm a man can store, and there is also a limit to his excitement. Under normal circumstances, after ejaculation, he will continue to maintain a certain level of excitement, and the penis will still be engorged with blood, so it will remain hard. However, after some people ejaculate multiple times, their excitement has decreased and they are not as sensitive. They may become soft after ejaculation. But if it is not very hard after one time, it means that there is still something wrong with the body.

Ejaculation, a basic biological term, is the reflex action of male animals to eject semen during sexual behavior. For humans, ejaculation is a series of coordinated actions of various parts of the reproductive system, and semen is ejected from the penis.

It consists of a two-step spinal reflex, the primary of which is centered in the lumbosacral spinal cord, with sensory impulses transmitted by touch receptors in the glans penis. The neurophysiology of ejaculation is only partially understood. Studies have shown that dopaminergic systems promote ejaculation while serotonergic systems inhibit ejaculation.

The primary neural mechanism of ejaculation is somatic innervation, but it also involves nerves and fibers from the autonomic nervous system in the vagina. The autonomic branches of the parasympathetic nervous system primarily control erectile function, while the sympathetic and somatic branches primarily control emission and ejaculation.

"Impotence" is the former name of "erectile dysfunction (ED)". In 1992, after discussion by relevant experts, the National Institutes of Health of the United States decided to replace the term impotence with erectile dysfunction. Erectile dysfunction (ED) refers to the persistent inability of the penis to achieve and maintain an erection sufficient for satisfactory sexual intercourse in the past three months; ED is one of the most common sexual dysfunctions in men. Although ED is not a life-threatening disease, it is closely related to the patient's quality of life, sexual partner relationship, and family stability, and is also an early warning sign of many physical diseases.

The occurrence of ED is not only affected by physical diseases such as age, cardiovascular disease, diabetes and hyperlipidemia, as well as psychological and environmental factors such as sexual partner relationships and home conditions, but also by bad living habits, drugs, surgery, race, culture, religion and socioeconomic factors.

1. Age

Current research suggests that age is the strongest independent factor among ED-related risk factors. As age increases, the serum androgen level decreases significantly, which may be the direct cause, but there is no research result proving that there is a significant relationship between the decrease in serum free testosterone and ED. In addition, as age increases, the structure of the penis tunica albuginea and corpus cavernosum changes, which may lead to a decrease in venous blood return capacity, an increase in the prevalence of cardiovascular and cerebrovascular diseases, hypertension, diabetes, and the treatment of these diseases, all of which damage the penis' erectile function to varying degrees, and this trend also increases with age.

2. Cardiovascular disease

Cardiovascular disease is the main somatic disease associated with ED, including atherosclerosis, peripheral vascular disease, hypertension and myocardial infarction. Cardiovascular disease causes arterial ED by affecting the arterial blood supply to the corpus cavernosum. Some studies have also found that ED may be the primary manifestation or "warning signal" of cardiovascular disease.

3. Diabetes

Diabetes can affect erectile function by affecting the autonomic nervous system, peripheral vascular system and psychoneural system. The severity and prevalence of ED are significantly related to factors such as age of diabetes, type of diabetes, blood sugar control, diabetic neuropathy, diabetic nephropathy and hypertension.

4. Abnormal blood lipid metabolism

The role of hypercholesterolemia in sexual dysfunction is controversial. One study showed that men with total cholesterol (TC) greater than 240 mg/dl had a 1.83 times higher risk of ED than men with TC less than 180 mg/dl. In the MMAS (Massachusetts Aging Male Study), high-density lipoprotein was negatively correlated with the prevalence of ED.

5. Chronic prostatitis

Some patients with chronic prostatitis have symptoms such as premature ejaculation, loss of libido, erectile dysfunction and ejaculation pain. The mechanism of sexual dysfunction caused by chronic prostatitis is unclear. Most scholars believe that anxiety, depression, inferiority, loss of energy, fatigue, suspicion and insomnia are the main causes. The recurrence and non-healing of long-term testicular swelling and pain, perineum and penis discomfort, lower urinary tract symptoms, etc. also increase the psychological burden of patients. Since the sexual dysfunction of most patients with chronic prostatitis is caused by psychological factors, in addition to drug treatment, more psychological counseling and treatment are needed.